Pharma M2
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Natural adrenocortical hormones are produced in released by:
Hormonal steroids most influential on intermediary metabolism:
Glucocorticoid effect(s), EXCEPT
Example of short-to medium-acting glucocorticoid
Long-acting glucocorticoid
inhibition of 11 beta-hydroxylase activity (P450c11) would reduce formation of, EXCEPT
inhibition of 21 beta-hydroxylase activity (P450c21) would reduce formation of:
Some asthmatic patients exhibit aspirin sensitivity (and sensitivity to other NSAIDS). Daily administration of aspirin causes desensitization not only to aspirin also to other NSAIDS. This phenomenon is:
A 38 year old asthmatic who has been using beta adrenergic agonists for several years began to experience arrhythmias, premature atrial contractions. In order to suppress the arrhythmias, high-dose metoprolol therapy was started. Suggest a likely consequence:
Anti-asthma agent that acts in part by activating pulmonary beta-receptors thus increasing cAMP
An asthmatic is prescribed an adrenergic agent. After administration, the drug makes the patient tachycardiac and nervous. This drug was probably:
V/Q (ventilation/perfusion) mismatching which occurs in severe asthma results in hypoxemia. Terbutaline inhalation inproves the V/Q ratio. Why?
Corticosteroids and the treatment of asthma:
H2 Blocker most likely to inhibit P450 drug metabolizing system:
H2 blocker most likely to cause gynecomastia:
Example of second-generation antihistamine:
The most important pathophysiologic mechanism of mast cell and basophil histamine release:
Non-neuronal site(s) of histamine storage and release:
Histamine aerosol: effects on pulmonary function --
Most likely to reduce blood pressure by directly decreasing heart rate:
From the point of view of Starling's law which antihypertensive would be most likely to reduce contractility.
Negative inotropism
Increases pulmonary congestion in congestive heart failure (CHF)
Major neurotransmitter released at end organ effectors of the thoracolumbar division of the autonomic nervous system:
Methoxamine (Vasoxyl)-induced bradycardia would be prevented by:
Dopamine beta hydroxylase catalyzes:
Most potent at beta adrenergic receptors
alpha-2 receptor agonist; peripheral sympathomimetic
Inhibits neurotransmitter enzymic degradation:
Cardiac effects not like to be directly affected by the presence of an anticholinesterase:
Pilocarpine (Pilocar):
Interferes with norepinephrine release:
Atropine effects:
Cholinergic agent least likely to enter the brain:
Reversible, noncovalent inhibitor of acetylcholinesterase
Ganglionic blocker:
Predominant autonomic tone:
Powerful agonist at both alpha and beta adrenergic receptors
Positive inotropic drug that at low doses specifically promotes an increase in renal blood flow:
Antihypertensive effect due to activation of CNS alpha-2 receptors
